OnCo

B7-1 is a molecule on antigen-presenting cells that gives T cells their go signal through CD28, but it also feeds the CTLA-4 brake and can pair with PD-L1. Anti-PD-L1 antibodies such as sugemalimab and adebrelimab are built to stop PD-L1 binding both PD-1 and B7-1. This dossier gathers the 0 products (0 approved), 0 trials, 0 pathways and 0 resistance routes in the corpus that involve it, with external identifiers so it can be joined to UniProt, ChEMBL, Open Targets and the rest of biology.

Biology

The same ligand serves an accelerator (CD28) and a brake (CTLA-4), which is why CTLA-4 blockade increases T-cell priming. PD-L1 antibodies that also free B7-1 restore CD28 costimulation as well as lifting the PD-1 brake.

Where it is found
  • Dendritic cells, macrophages and activated B cells
  • Small-cell lung cancer (adebrelimab) and NSCLC and peripheral T-cell lymphoma (sugemalimab) populations
Class: checkpoint · Gene: CD80 · Facts checked 2026-09-22 · Target page

External identifiers

Built from HGNC, Ensembl, UniProt and ChEMBL ids

Products by modality and phase

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Resistance routes

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No pathway diagram carries this target as a node.

Companion diagnostics

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Preclinical models

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No model entry for this target yet; check the cancer entries on the models page.

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Literature

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Query for this target: (TITLE:"CD80" OR ABSTRACT:"CD80" OR TITLE:"B7-1" OR ABSTRACT:"B7-1" OR TITLE:"B7.1" OR ABSTRACT:"B7.1" OR TITLE:"CD28LG" OR ABSTRACT:"CD28LG" OR TITLE:"CD80 molecule" OR ABSTRACT:"CD80 molecule") AND (cancer OR tumor OR tumour OR oncology OR carcinoma OR lymphoma OR leukemia OR leukaemia OR myeloma OR sarcoma OR melanoma OR glioma). Results are unfiltered search hits about CD80 (B7-1), not a curated reading list.

Export

The dossier as machine-readable JSON: identifiers from HGNC, Ensembl, UniProt and ChEMBL, products with status, trials, pathways, hotspots, open questions and assays. The full entity record is in the open API at /api/v1/entities/cd80.json. Licence CC BY-NC 4.0.