OnCo

CD19 is a marker on B cells and B-cell cancers, and was the target of the first CAR-T therapies ever approved. This dossier gathers the 8 products (8 approved), 13 trials, 2 pathways and 3 resistance routes in the corpus that involve it, with external identifiers so it can be joined to UniProt, ChEMBL, Open Targets and the rest of biology.

Biology

B-cell co-receptor; loss of CD19 is a common escape mechanism after CAR-T.

Where it is found
  • ALL
  • DLBCL
  • Follicular lymphoma
  • Mantle cell lymphoma
  • CLL
Class: surface antigen · Gene: CD19 · Facts checked 2026-09-04 · Target page

Elsewhere: identifiers and databases

Built from HGNC, Ensembl, UniProt and ChEMBL ids

How common it is, by cancer

Full matrix →
CancerPrevalenceSource
Acute lymphoblastic leukaemia
>95%
Wikipedia
Diffuse large B-cell lymphoma
>95%
Wikipedia
Chronic lymphocytic leukaemia
>95%
Wikipedia

Approximate, population-level figures; the measure column says what was counted. Ranges show the midpoint as a bar.

Products by modality and phase

Browse products →
TrialPhaseStatus
frontMIND
NCT04824092
3Positive
COG AALL1731
NCT03914625
3Positive
ECOG-ACRIN E1910
NCT02003222
3Positive
TRANSFORM
NCT03575351
3Positive
ZUMA-7
NCT03391466
3Positive
Interfant-06
NCT00550992
3Mixed
TOWER
NCT02013167
3Positive
LOTIS-2
NCT03589469
2Positive
D-ALBA (GIMEMA LAL2116)
NCT02744768
2Positive
L-MIND
NCT02399085
2Positive
ELIANA
NCT02435849
2Positive
FELIX
NCT04404660
1/2Positive
TRANSCEND CLL 004
NCT03331198
1/2Positive

Resistance routes that involve this target

Unaddressed routes →
Antigen loss · CD19 CAR-T
CD19-negative relapse
Frequency: ~30–50% of relapses in ALL, less in lymphoma

Alternative splicing, mutation, or lineage switch (to myeloid) removes the CD19 epitope.

Countermeasures · 1
Pharmacology · CD19 CAR-T
CD19-positive relapse from poor CAR-T persistence

Limited expansion or early loss of CAR-T cells; 4-1BB products persist longer than CD28.

Countermeasures · 1
Immune evasion · CD19 CAR-T
Immunosuppressive microenvironment and T-cell exhaustion

PD-1 upregulation, TGF-β, and myeloid suppression in lymphoma.

Countermeasures · 1

Pathways where it is a node

Pathway-to-drug matrix →
  • Resistance routes: how a blocked pathway comes back
    Node: 5 Efflux, sanctuary, antigen loss · 6 druggable nodes

    When a drug blocks a cancer's engine, the cancer has five ways back: change the part the drug binds, make more of it, take a side road, switch to a different engine altogether, or stop letting the drug in. Knowing which route a tumour took decides the next drug.

    Which nodes have drugs →
  • T-cell exhaustion
    Node: CAR-T exhaustion · 3 druggable nodes

    T cells that see their target for weeks on end without winning gradually shut down: they raise a set of brakes (PD-1, LAG-3, TIM-3, TIGIT), lose their ability to kill, and eventually lock this state into their DNA. Checkpoint drugs rescue the ones that are only partly exhausted; the terminally exhausted are beyond reach.

    Which nodes have drugs →

Companion diagnostics and assays

Assay registry →

No companion diagnostic in the registry measures this target.

Preclinical models

All models →
Cell lineIdentifiersWhy it is used
NALM-6CVCL_0092 · ACH-000938Pre-B ALL; CAR-T potency standard.
RajiCVCL_0511 · ACH-000654Burkitt; CD19 and CD20.
DaudiCVCL_0008 · ACH-000786Burkitt; beta-2-microglobulin null (no HLA class I), useful for MHC-independent killing.
REHCVCL_1650 · ACH-000960Pre-B ALL.
JeKo-1CVCL_1865 · ACH-000357Mantle-cell lymphoma.
K-562 CD19not resolvedCD19-transduced K-562 as an artificial target with CD19-negative parent as control.
  1. 01

    Can in vivo CAR-T (lentiviral or mRNA delivery of the CAR to the patient's own T cells) match ex vivo products in B-cell malignancies?

    translationalindustry

    Why unresolved. Manufacturing cost, slot limits and vein-to-vein time are the main barriers to CD19 CAR-T access; in vivo approaches remove them but first-in-human data are only now emerging.

    What would answer it. Phase 1/2 data showing comparable expansion, persistence and complete-response rates, then a randomised comparison with an approved product.

    Source: ZUMA-1, NEJM 2017

Ideas and companies

Key papers and the live literature

Preprints →
Latest papers · live from Europe PMC
Open in Europe PMC

Query for this target: (TITLE:"CD19" OR ABSTRACT:"CD19") AND (cancer OR tumor OR tumour OR oncology OR carcinoma OR lymphoma OR leukemia OR leukaemia OR myeloma OR sarcoma OR melanoma OR glioma). Results are unfiltered search hits about CD19, not a curated reading list.

Export

The dossier as machine-readable JSON: identifiers from HGNC, Ensembl, UniProt and ChEMBL, products with status, trials, pathways, hotspots, open questions and assays. The full entity record is in the open API at /api/v1/entities/cd19.json. Licence CC BY 4.0.